Download PDF by Martin Kaltenbach MD, Ronald E. Vlietstra MB, ChB (auth.): Concise Cardiology

By Martin Kaltenbach MD, Ronald E. Vlietstra MB, ChB (auth.)

ISBN-10: 0387913947

ISBN-13: 9780387913940

ISBN-10: 3662307634

ISBN-13: 9783662307632

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Figure 22 features typically deformed dye dilution curves as a result of right-to-left or left-to-right shunts. It also shows that the area under the curve increases with decreasing cardiac output. The Fick cardiac output is calculated from the oxygen uptake and arteriovenous difference, using the equation CO (l/min) == oxygen uptake (ml/min) A-V02 difference (ml/lOO ml) x 10 The indicator dye dilution cardiac output uses the equation CO (l/min) == _I- x ~ . t 1000 I == dye dose (mg), c == mean concentration (mg/ml), t == duration of unrecirculated dye curve.

6 Implications of coronary heart disease, risk factors, progression Cigarette smoking is a well-documented risk factor. The frequency of myocardial infarction and angina pectoris correlates with the number of cigarettes smoked per day. A clear lower limit, that is an amount of cigarettes which is not dangerous, does not exist. The risk factor of cigarette smoking is most important in young men and women, less so in persons above 60 years of age, because then age as a risk factor per se becomes of dominant importance.

Wit hout 0 coronary arteriosclerosis Fig. 33. Age and sex· ad justed comparison of patients with angiographically documented coronary artery disease with patients having normal coronary arteries. There is a small but significant correlation of disease with elevated triglycerides, with higher total and LDL cholesterol, and decreased HDL cholesterol. ) HDL Cholesterol LDL HDL 19 HDL LDL Triglyceride Cholesterol - 30 -20 -1 0 0 +10 +20 +30 % Fig. 34. Compared to healthy persons the average cholesterol is elevated by 4%, triglyceride by 28%, and LDL by 6% in persons with coronary artery disease, and HDL is 15% lower.

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Concise Cardiology by Martin Kaltenbach MD, Ronald E. Vlietstra MB, ChB (auth.)


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