By Alan M Jones (auth.), Richard M. Amasino (eds.)
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Soc. Trans. 20:89-92. McCarty, D. R, C. B. Carson, M. Lazar and S. C. Simonds. 1989a. Transposable element induced mutations of the viviparous-l gene of maize. Dev. Genetics. 10: 473-481. McCarty, D. R, C. B. Carson, P. S. Stinard and D. S. Robertson. 1989b. Molecular analysis of viviparous-I: An abscisic acid insensitive mutant of maize. Plant Cell 1: 523-532. McCarty, D. R and C. B. Carson. 1990. The molecular genetics of seed maturation in maize. Physiol Plant. 81: 267-272. McCarty, D. R, T.
Brooker JD, Russell DW (1979) Regulation of microsomal 3-hydroxy-3methylglutaryl coenzyme A reductase from pea seedlings: rapid posttranslational phytochrome-mediated decrease in activity and in vivo regulation by isoprenoid products. Arch Biochem Biophys 198: 323-334 8. Brown MS, Goldstein JL (1980) Multivalent feedback regulation of HMG CoA reductase, a control mechanism coordinating isoprenoid synthesis and cell growth. J Lipid Res 21: 505-517 9. Goldstein JL, Brown MS (1990) Regulation of the mevalonate pathway.
Accordingly, we examined the 40 effects of various cytokinins on growth of lovastatin-treated tobacco cells. M lovastatin, indicating that growth inhibition at this concentration of lovastatin was caused, at least in part, by reduced endogenous synthesis of cytokinin or other essential isoprenoid(s) that can be synthesized from zeatin. M lovastatin, arguing against the latter (these compounds are not isoprenoids). M, suggesting that lovastatin either inhibits the biosynthesis of other essential isoprenoids or has non-specific, toxic effects at high concentrations (data not shown).
Cellular Communication in Plants by Alan M Jones (auth.), Richard M. Amasino (eds.)